Can erythrocytes release biologically active NO? | Cell Communication and Signaling | Full Text
Assessing Sediment Nutrient Storage and Release in California's Sacramento-San Joaquin Delta | U.S. Geological Survey
-Immunohistochemistry-Paraffin-NBP1-51582-img0002.jpg "eNOS Antibody (6H2) - BSA Free (NBP1-51582): Novus Biologicals")
eNOS Antibody (6H2) - BSA Free (NBP1-51582): Novus Biologicals
Ac2-26 ameliorates lung ischemia-reperfusion injury via the eNOS pathway - ScienceDirect
Antioxidants | Free Full-Text | Endogenous and Exogenous Antioxidants in Skeletal Muscle Fatigue Development during Exercise
eNOS alleles do not rescue impaired growth, proliferation, or litter... | Download Scientific Diagram
IJMS | Free Full-Text | Ginsenoside Rb1 Blocks Ritonavir-Induced Oxidative Stress and eNOS Downregulation through Activation of Estrogen Receptor-Beta and Upregulation of SOD in Human Endothelial Cells
Regulation of endothelium-derived nitric oxide production by the protein kinase Akt | Nature
Zeb1-Hdac2-eNOS circuitry identifies early cardiovascular precursors in naive mouse embryonic stem cells | Nature Communications
Targeting mechanosensitive endothelial TXNDC5 to stabilize eNOS and reduce atherosclerosis in vivo | Science Advances
Role of glutathione biosynthesis in endothelial dysfunction and fibrosis - ScienceDirect
Expression of CD70 Modulates Nitric Oxide and Redox Status in Endothelial Cells | Arteriosclerosis, Thrombosis, and Vascular Biology
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Frontiers | A20/TNFAIP3 Increases ENOS Expression in an ERK5/KLF2-Dependent Manner to Support Endothelial Cell Health in the Face of Inflammation
eNOS phosphorylation: A pivotal molecular switch in vasodilation and cardioprotection? - Journal of Molecular and Cellular Cardiology
S-glutathionylation uncouples eNOS and regulates its cellular and vascular function | Nature
eNOS and stroke: prevention, treatment and recovery | Future Neurology
Coordinated Control of Endothelial Nitric-oxide Synthase Phosphorylation by Protein Kinase C and the cAMP-dependent Protein Kinase - ScienceDirect
Biomolecules | Free Full-Text | Nitric Oxide-Dependent Pathways as Critical Factors in the Consequences and Recovery after Brain Ischemic Hypoxia
